Lipopolysaccharide-responder and nonresponder C3H mouse strains are equally susceptible to an induced Escherichia coli urinary tract infection.

Abstract

Host defense against bacterial urinary tract infections (UTI) includes both inflammatory and immune responses to infecting bacteria. The cellular events leading up to local inflammation are thought to be under genetic control and initiated by lipopolysaccharides (LPS) of gram-negative bacteria such as Escherichia coli. It has been previously reported that… (More)

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@article{Hopkins1996LipopolysaccharideresponderAN, title={Lipopolysaccharide-responder and nonresponder C3H mouse strains are equally susceptible to an induced Escherichia coli urinary tract infection.}, author={William D. Hopkins and Annette P Gendron-Fitzpatrick and Dan McCarthy and James Edward Haine and David T. Uehling}, journal={Infection and immunity}, year={1996}, volume={64 4}, pages={1369-72} }