Leukocyte-endothelial cell interactions in chronic vasospasm after subarachnoid hemorrhage

@article{Gallia2006LeukocyteendothelialCI,
  title={Leukocyte-endothelial cell interactions in chronic vasospasm after subarachnoid hemorrhage},
  author={G. Gallia and R. Tamargo},
  journal={Neurological Research},
  year={2006},
  volume={28},
  pages={750 - 758}
}
  • G. Gallia, R. Tamargo
  • Published 2006
  • Medicine
  • Neurological Research
    • Abstract Leukocyte-endothelial cell interactions appear to be the root cause of chronic vasospasm after aneurysmal subarachnoid hemorrhage (aSAH). Early clinical observations and indirect experimental evidence suggested an association between inflammation and chronic vasospasm. Early clinical observations in patients with post-hemorrhagic vasospasm included pyrexia, leukocytosis and the presence of circulating immune complexes. Inflammatory infiltrates and increased levels of immunoglobulins… CONTINUE READING
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    References

    SHOWING 1-10 OF 112 REFERENCES
    Prevention of cerebral vasospasm by a humanized anti-CD11/CD18 monoclonal antibody administered after experimental subarachnoid hemorrhage in nonhuman primates.
    • 73
    • PDF
    Monoclonal antibodies against ICAM-1 and CD18 attenuate cerebral vasospasm after experimental subarachnoid hemorrhage in rabbits.
    • 140
    • PDF
    Cerebrovascular inflammation following subarachnoid hemorrhage.
    • 171
    • PDF
    Endothelial cell expression of intercellular adhesion molecule 1 in experimental posthemorrhagic vasospasm.
    • 74
    Prevention of vasospasm by anti-CD11/CD18 monoclonal antibody therapy following subarachnoid hemorrhage in rabbits.
    • 50
    Rise in serum soluble intercellular adhesion molecule-1 levels with vasospasm following aneurysmal subarachnoid hemorrhage.
    • 59
    • PDF
    Monoclonal antibody against E selectin attenuates subarachnoid hemorrhage-induced cerebral vasospasm.
    • 61
    The role of inflammation in experimental cerebral vasospasm.
    • 150
    Inflammation in stroke and focal cerebral ischemia.
    • 529
    Inhibition of vasospasm with lymphocyte function-associated antigen-1 monoclonal antibody in a femoral artery model in rats.
    • 49
    • PDF