Japanese encephalitis virus infection decrease endogenous IL-10 production: correlation with microglial activation and neuronal death.

@article{Swarup2007JapaneseEV,
  title={Japanese encephalitis virus infection decrease endogenous IL-10 production: correlation with microglial activation and neuronal death.},
  author={Vivek Swarup and Joydeep Ghosh and Rachna Duseja and Soumya Sankar Ghosh and Anirban Basu},
  journal={Neuroscience letters},
  year={2007},
  volume={420 2},
  pages={144-9}
}
The anti-inflammatory cytokine interleukin (IL)-10 is synthesized in the central nervous system (CNS) and acts to limit clinical symptoms of stroke, multiple sclerosis, Alzheimer's disease, meningitis, and the behavioral changes that occur during bacterial infections. Expression of IL-10 is critical during the course of most major diseases in the CNS and promotes survival of neurons and all glial cells in the brain by blocking the effects of proinflammatory cytokines and by promoting expression… CONTINUE READING
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