Intravenous lidocaine for effective pain relief after bimaxillary surgery

@article{Lee2017IntravenousLF,
  title={Intravenous lidocaine for effective pain relief after bimaxillary surgery},
  author={Uilyong Lee and Young-Jun Choi and Geun Joo Choi and Hyun Kang},
  journal={Clinical Oral Investigations},
  year={2017},
  volume={21},
  pages={2645-2652}
}
ObjectivesThe aim of this prospective, randomized, double-blind, placebo-controlled study was to evaluate the analgesic effect of intravenous lidocaine on postoperative pain in bimaxillary surgery.Materials and methodsBetween July 2015 and November 2015, 52 consecutive patients that underwent bimaxillary surgery were recruited to the present study. The patients were randomly divided into two groups: group L (1.5 mg/kg bolus and 2 mg/kg/h continuous infusion during the operation) and group C… 
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The effects (benefits and risks) of perioperative intravenous (IV) lidocaine infusion compared to placebo/no treatment or compared to epidural analgesia on postoperative pain and recovery in adults undergoing various surgical procedures are assessed.
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The aim of this randomized, double‐blinded, controlled trial was to investigate the effect of perioperative lidocaine infusion on postoperative early recovery quality in upper airway surgery.
Pain Medicine in Oral and Maxillofacial Surgery
A significant amount of empirical progress has been made in the management of pain over the last century,largely as a result of the introduction of a more effective pharmacological agent and the
Management of postoperative pain in maxillofacial surgery.
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The evidence base for postoperative analgesia after maxillofacial surgery is described and the implications of poorly managed pain, risk factors for the development of severe pain, and pharmacological and non-pharmacological analgesic strategies to manage it are discussed.
Lidocaine attenuates lipopolysaccharide-induced inflammatory responses and protects against endotoxemia in mice by suppressing HIF1α-induced glycolysis.
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Lidocaine can be used as a potential therapeutic agent for sepsis because it dose-dependently inhibits lipopolysaccharide (LPS)-induced production of tumor necrosis factor-alpha (TNF-α) and interleukin-6 (IL-6) in macrophages and that lidocaine protects mice from LPS-induced inflammation.

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