Insulin restores L-arginine transport requiring adenosine receptors activation in umbilical vein endothelium from late-onset preeclampsia.

@article{Salsoso2015InsulinRL,
  title={Insulin restores L-arginine transport requiring adenosine receptors activation in umbilical vein endothelium from late-onset preeclampsia.},
  author={Roc{\'i}o Salsoso and Enrique Guzm{\'a}n-Gutierrez and T Estefan{\'i}a Saez and Katherinne Bugue{\~n}o and Marco A. Leyva Ram{\'i}rez and Marcelo Far{\'i}as and F. Mart{\'i}nez Pardo and Andrea Leiva and Cristina Sanhueza and Alfonso Mate and Carlos V{\'a}zquez and Luis Sobrevia},
  journal={Placenta},
  year={2015},
  volume={36 3},
  pages={
          287-96
        }
}
INTRODUCTION Preeclampsia is associated with impaired placental vasodilation and reduced endothelial nitric oxide synthase (eNOS) activity in the foetoplacental circulation. Adenosine and insulin stimulate vasodilation in endothelial cells, and this activity is mediated by adenosine receptor activation in uncomplicated pregnancies; however, this activity has yet to be examined in preeclampsia. Early onset preeclampsia is associated with severe placental vasculature alterations that lead to… CONTINUE READING
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