Inhibition of nitric oxide synthesis accelerates the recovery of polysynapitc reflex potentials after transient spinal cord ischemia in cats

@article{Nemoto1997InhibitionON,
  title={Inhibition of nitric oxide synthesis accelerates the recovery of polysynapitc reflex potentials after transient spinal cord ischemia in cats},
  author={Tetsuharu Nemoto and Toshihiko Sekikawa and Toshio Suzuki and Hideshige Moriya and Haruaki Nakaya},
  journal={Naunyn-Schmiedeberg's Archives of Pharmacology},
  year={1997},
  volume={355},
  pages={447-451}
}
Nitric Oxide (NO) has been implicated as a mediator of neuronal injury in vascular stroke. On the other hand, NO is suggested to play a neuroprotective role by increasing blood flow during cerebral ischemia. In order to evaluate the role of NO in the spinal cord ischemia, effects of nitric oxide synthase (NOS) inhibition on the recovery of reflex potentials after a transient spinal cord ischemia were examined in urethane-chloralose anesthetized spinal cats. Spinal cord ischemia was produced by… CONTINUE READING

References

Publications referenced by this paper.
Showing 1-10 of 25 references

Effects of nicorandil on the recovery of reflex potentials after spinal cord ischaemia in cats

T Suzuki, T Sekikawa, T Nemoto, H Moriya, H Nakaya
Br J Pharmacol • 1995

Nitric oxide measured by a porphyrinic microsensor in rat brain after transient middle cerebral artery occlusion.

Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism • 1993
View 2 Excerpts

The L-arginine-nitric oxide pathway.

The New England journal of medicine • 1993
View 1 Excerpt

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