Inhibition of experimental asthma by indoleamine 2,3-dioxygenase.

@article{Hayashi2004InhibitionOE,
  title={Inhibition of experimental asthma by indoleamine 2,3-dioxygenase.},
  author={Tomoko Hayashi and L. Beck and C. Rossetto and Xing-mu Gong and O. Takikawa and K. Takabayashi and D. Broide and D. Carson and E. Raz},
  journal={The Journal of clinical investigation},
  year={2004},
  volume={114 2},
  pages={
          270-9
        }
}
Epidemiological evidence points to the inverse relationship between microbial exposure and the prevalence of allergic asthma and autoimmune diseases in Westernized countries. The molecular basis for this observation has not yet been completely delineated. Here we report that the administration of certain toll-like receptor (TLR) ligands, via the activation of innate immunity, induces high levels of indoleamine 2,3-dioxygenase (IDO), the rate-limiting enzyme of tryptophan catabolism in various… Expand
Indoleamine-2,3-dioxygenase modulation of allergic immune responses
The indoleamine 2,3‐dioxygenase (IDO) pathway controls allergy
Inhibition of acute lethal pulmonary inflammation by the IDO–AhR pathway
Indoleamine 2,3-dioxygenase in lung dendritic cells promotes Th2 responses and allergic inflammation
Molecules in focus: indoleamine 2,3-dioxygenase.
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CDllc+ cells modulate pulmonary immune responses by production of indoleamine 2,3-dioxygenase.
Enhancement of Innate Immunity againstMycobacterium avium Infection by Immunostimulatory DNA Is Mediated by Indoleamine 2,3-Dioxygenase
  • Tomoko Hayashi, Savita P. Rao, +7 authors Eyal Raz
  • Biology, Medicine
  • Infection and Immunity
  • 2001
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