• Corpus ID: 89325847

Inhibition of Extracellular Ca2 Entry by YC-1, an Activator of Soluble Guanylyl Cyclase, through a Cyclic GMP-independent Pathway in Rat Neutrophils

@inproceedings{2001InhibitionOE,
  title={Inhibition of Extracellular Ca2 Entry by YC-1, an Activator of Soluble Guanylyl Cyclase, through a Cyclic GMP-independent Pathway in Rat Neutrophils},
  author={王繼平 and 黃麗嬌 and 郭盛助},
  year={2001}
}

Stimulation of cellular free Ca2+ elevation and inhibition of store-operated Ca2+ entry by kazinol B in neutrophils

TLDR
The results indicate that kazinol B stimulates Ca2+ release from internal Ca 2+ store and activates external Ca2- influx mainly through a non-store-operated Ca2+.

Vasorelaxing Effect of BAY 41-2272 in Rat Basilar Artery: Involvement of cGMP-Dependent and Independent Mechanisms

TLDR
BAY 41-2272 potently relaxes the rat basilar artery in a synergistic fashion with NO andTargeting the sGC with selective activators, such as BAY 41,2272, may represent a new therapy to treat cerebrovascular disease.

MECHANISMS UNDERLYING RELAXATION OF RABBIT AORTA BY BAY 41‐2272, A NITRIC OXIDE‐INDEPENDENT SOLUBLE GUANYLATE CYCLASE ACTIVATOR

TLDR
The mechanisms underlying the relaxant effect of BAY 41‐2272 in endothelium‐intact and ‐denuded precontracted rabbit aortic rings were investigated.

Toborinone and olprinone, phosphodiesterase III inhibitors, inhibit human platelet aggregation due to the inhibition of both calcium release from intracellular stores and calcium entry

TLDR
Toborinone and olprinone inhibit human platelet aggregation, though these concentrations are higher than their therapeutic plasma concentrations, and the inhibitory effects of both drugs are related to the inhibition of bothCa2+ release and Ca2+ entry through [cAMP]i elevation.

2-Benzyloxybenzaldehyde inhibits formyl peptide-stimulated increase in intracellular Ca2+ in neutrophils mainly by blocking Ca2+ entry

TLDR
CCY1a blocks fMLP-induced Ca2+ entry into neutrophils probably by blocking the relevant Ca2+.