Influence of Hyperhomocysteinemia on the Cellular Redox State – Impact on Homocysteine-Induced Endothelial Dysfunction

@inproceedings{Weiss2003InfluenceOH,
  title={Influence of Hyperhomocysteinemia on the Cellular Redox State – Impact on Homocysteine-Induced Endothelial Dysfunction},
  author={N. Weiss and S. Heydrick and O. Postea and C. Keller and J. Keaney and J. Loscalzo},
  booktitle={Clinical chemistry and laboratory medicine},
  year={2003}
}
Abstract Hyperhomocysteinemia is an independent risk factor for the development of atherosclerosis. An increasing body of evidence has implicated oxidative stress as being contributory to homocysteine's deleterious effects on the vasculature. Elevated levels of homocysteine may lead to increased generation of superoxide by a biochemical mechanism involving nitric oxide synthase, and, to a lesser extent, by an increase in the chemical oxidation of homocysteine and other aminothiols in the… Expand
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