Increased vagal cardiac nerve traffic prolongs ventricular refractoriness in patients undergoing electrophysiology testing.

@article{Ellenbogen1990IncreasedVC,
  title={Increased vagal cardiac nerve traffic prolongs ventricular refractoriness in patients undergoing electrophysiology testing.},
  author={Kenneth A. Ellenbogen and Michael L. Smith and Dwain L Eckberg},
  journal={The American journal of cardiology},
  year={1990},
  volume={65 20},
  pages={1345-50}
}
Stimulation of the vagus nerve in animals causes prolongation of sinus cycle length, atrioventricular nodal conduction and ventricular refractoriness. Vagal stimulation appears to have a protective effect in animal models of sudden death. The electrophysiologic effects of enhanced vagal activity on right ventricular (RV) refractoriness in man have not been studied previously. The comparative effects of enhanced vagal tone (neck suction to -60 mm Hg) on sinus cycle length and RV refractoriness… CONTINUE READING