Increased interleukin-10 and interferon-γ levels in Plasmodium vivax malaria suggest a reciprocal regulation which is not altered by IL-10 gene promoter polymorphism

@inproceedings{Medina2011IncreasedIA,
  title={Increased interleukin-10 and interferon-γ levels in Plasmodium vivax malaria suggest a reciprocal regulation which is not altered by IL-10 gene promoter polymorphism},
  author={Tiago da S. Medina and Sheyla P. T. Costa and Maria D. L. Oliveira and Ana Maria Revor{\^e}do da Silva Ventura and Jos{\'e} Souza and T{\'a}ssia Fernanda Furo Gomes and Antonio Carlos Ros{\'a}rio Vallinoto and Marinete Marins P{\'o}voa and Jo{\~a}o S. Silva and Maristela G Cunha},
  booktitle={Malaria Journal},
  year={2011}
}
In human malaria, the naturally-acquired immune response can result in either the elimination of the parasite or a persistent response mediated by cytokines that leads to immunopathology. The cytokines are responsible for all the symptoms, pathological alterations and the outcome of the infection depends on the reciprocal regulation of the pro and anti-inflammatory cytokines. IL-10 and IFN-gamma are able to mediate this process and their production can be affected by single nucleotide… CONTINUE READING
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