Increased AS 160 phosphorylation , but not TBC 1 D 1 phosphorylation , with increased postexercise insulin sensitivity in rat skeletal muscle

@inproceedings{Funai2009IncreasedA1,
  title={Increased AS 160 phosphorylation , but not TBC 1 D 1 phosphorylation , with increased postexercise insulin sensitivity in rat skeletal muscle},
  author={Katsuhiko Funai and George G. Schweitzer and Naveen Sharma and Makoto Kanzaki and Gregory D. Cartee},
  year={2009}
}
Funai K, Schweitzer GG, Sharma N, Kanzaki M, Cartee GD. Increased AS160 phosphorylation, but not TBC1D1 phosphorylation, with increased postexercise insulin sensitivity in rat skeletal muscle. Am J Physiol Endocrinol Metab 297: E242–E251, 2009. First published May 12, 2009; doi:10.1152/ajpendo.00194.2009.—A single exercise bout can increase insulin-independent glucose transport immediately postexercise and insulin-dependent glucose transport (GT) for several hours postexercise. Akt substrate of… CONTINUE READING

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Prolonged increase in insulin-stimulated glucose trans E249 AS160, TBC1D1, AND POSTEXERCISE INSULIN SENSITIVITY AJP-Endocrinol Metab

  • GD Cartee, DA Young, MD Sleeper, J Zierath, H Wallberg-Henriksson, JO. Holloszy
  • N ovem er 16,
  • 2009
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