Inactivation of smad-transforming growth factor beta signaling by Ca(2+)-calmodulin-dependent protein kinase II.

Abstract

Members of the transforming growth factor beta (TGF-beta) family transduce signals through Smad proteins. Smad signaling can be regulated by the Ras/Erk/mitogen-activated protein pathway in response to receptor tyrosine kinase activation and the gamma interferon pathway and also by the functional interaction of Smad2 with Ca(2+)-calmodulin. Here we report… (More)

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@article{Wicks2000InactivationOS, title={Inactivation of smad-transforming growth factor beta signaling by Ca(2+)-calmodulin-dependent protein kinase II.}, author={Stephen J Wicks and Shuai Lui and Nadia Abdel-Wahab and Raina Mason and Andrew D. Chantry}, journal={Molecular and cellular biology}, year={2000}, volume={20 21}, pages={8103-11} }