In vivo biostability of shore 55D polyether polyurethanes with and without fluoropolymer surface modifying endgroups.

@article{Ward2006InVB,
  title={In vivo biostability of shore 55D polyether polyurethanes with and without fluoropolymer surface modifying endgroups.},
  author={Robert S Ward and James G. Anderson and Rick D. McVenes and Ken S Stokes},
  journal={Journal of biomedical materials research. Part A},
  year={2006},
  volume={79 4},
  pages={836-45}
}
Polyether polyurethanes are subject to autooxidation and environmental stress cracking (ESC) because of interactions with lysosomal oxygen-free radicals. Oxidation can also be catalyzed by and caused by direct (redox) reaction with transition metal ions (metal ion oxidation, MIO). The source of the ions is corrosion of metallic parts within an implanted device. A previous study on a Shore 80A polyether polyurethane modified with fluoropolymer surface modifying end groups demonstrated improved… CONTINUE READING