In utero and lactational exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) induces a reduction in epididymal and ejaculated sperm number in rhesus monkeys.

@article{Arima2009InUA,
  title={In utero and lactational exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) induces a reduction in epididymal and ejaculated sperm number in rhesus monkeys.},
  author={Akihiro Arima and Hirohito Kato and Yojiro Ooshima and Taishi Tateishi and Ayumi Inoue and Atsunobu Muneoka and Toshio Ihara and Shunichi Kamimura and Toshio Fukusato and Shunichiro Kubota and Hiroyuki Sumida and Mineo Yasuda},
  journal={Reproductive toxicology},
  year={2009},
  volume={28 4},
  pages={
          495-502
        }
}
A long-term developmental toxicity study of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) exposure was performed in rhesus monkeys and the effect on male reproductive organs was determined in the second generation. Dams received 0, 30 or 300 ng/kg TCDD subcutaneously on Day 20 of gestation, and then 5% of the initial dose was injected every 30 days until Day 90 after delivery. The offspring were maintained until reaching sexual maturity, and evaluated by semen analysis, and histopathology of the… CONTINUE READING
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