In my end is my beginning: control of end resection and DSBR pathway ‘choice’ by cyclin-dependent kinases

Abstract

The genome is constantly subjected to chemical alterations that have the potential to cause genetic mutation, chromosomal rearrangements and, in the case of multicellular organisms, cancer. Particular vulnerability exists during DNA replication, when the two DNA strands of a chromosome separate to form templates for the synthesis of sister chromatids… (More)
DOI: 10.1038/sj.onc.1208609

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Cite this paper

@article{Scully2005InME, title={In my end is my beginning: control of end resection and DSBR pathway ‘choice’ by cyclin-dependent kinases}, author={Ralph Scully and Anyong Xie}, journal={Oncogene}, year={2005}, volume={24}, pages={2871-2876} }