Impaired slow axonal transport in diabetic peripheral nerve is independent of RAGE.

@article{Juranek2013ImpairedSA,
  title={Impaired slow axonal transport in diabetic peripheral nerve is independent of RAGE.},
  author={Judyta Karolina Juranek and Matthew S. Geddis and Rosa H Rosario and Ann Marie Schmidt},
  journal={The European journal of neuroscience},
  year={2013},
  volume={38 8},
  pages={3159-68}
}
Diabetic peripheral nerve dysfunction is a common complication occurring in 30-50% of long-term diabetic patients. The pathogenesis of this dysfunction remains unclear but growing evidence suggests that it might be attributed, in part, to alteration in axonal transport. Our previous studies demonstrated that RAGE (Receptor for Advanced Glycation Endproducts) contributes to the pathogenesis of diabetic peripheral neuropathy and impairs nerve regeneration consequent to sciatic nerve crush… CONTINUE READING
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