Impaired exercise-induced vasodilatation in chronic atrial fibrillation--role of endothelium-derived nitric oxide.

@article{Takahashi2002ImpairedEV,
  title={Impaired exercise-induced vasodilatation in chronic atrial fibrillation--role of endothelium-derived nitric oxide.},
  author={Nobuyuki Takahashi and Yutaka Ishibashi and Toshio Shimada and Takeshi Sakane and Shuzo Ohata and Takashi Sugamori and Yoko Ohta and Shin-ichi Inoue and Ko Nakamura and Hiromi Shimizu and Harumi Katoh and Yo Murakami},
  journal={Circulation journal : official journal of the Japanese Circulation Society},
  year={2002},
  volume={66 6},
  pages={583-8}
}
Exercise capacity is often reduced in patients with atrial fibrillation (AF), but very few studies have focused on changes in endothelial function as a potential mechanism for the exercise limitation. The present study used using venous occlusion plethysmography to investigate whether nitric oxide (NO)-mediated vasodilatation is attenuated during exercise in patients with AF by measuring forearm blood flow (FBF) in 10 patients at rest and immediately after 2 levels of rhythmic handgrip exercise… CONTINUE READING
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