Idiopathic hypersomnia.

@article{Billiard1996IdiopathicH,
  title={Idiopathic hypersomnia.},
  author={Michel M. Billiard},
  journal={Neurologic clinics},
  year={1996},
  volume={14 3},
  pages={
          573-82
        }
}
Idiopathic hypersomnia is not as well delineated as narcolepsy and its history is much more recent. There are at least two forms of the disorder: (1) a polysymptomatic form, characterized by excessive daytime sleepiness, nocturnal sleep of abnormally long duration, and signs of sleep drunkenness on awakening, and (2) a monosymptomatic form that manifests only by excessive daytime sleepiness. The most widely used laboratory procedures are nocturnal polysomnographic recording following by an MSLT… Expand
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30
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87 Citations

Citation Type

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Idiopathic Hypersomnia.
TLDR
There are a number of cases of isolated excessive daytime sleepiness with no prolonged night sleep, no difficulty waking up, which lay between narcolepsy and genuine idiopathic hypersomnia and there is a definite need to further develop laboratory investigations to help identify and classify these cases. Expand
Idiopathic hypersomnia: a homogeneous or heterogeneous disease?
TLDR
IH patients with long sleep duration differences clinically as well as by objective measures at the time of diagnosis and in long-term follow up from IH patients without long 24-h sleep time represent an independent clinical entity to be considered in the revised ICSD-3 criteria. Expand
Idiopathic hypersomnia with and without long sleep time: a controlled series of 75 patients.
TLDR
Hypersomnia, especially with long sleep time, is frequently associated with evening chronotype and young age, and is inadequately diagnosed using MSLT. Expand
Update on the Treatment of Idiopathic Hypersomnia
TLDR
It is reasonable to start with modafinil as a first choice treatment for idiopathic hypersomnia, and methylphenidate and dexamphetamine are good alternatives. Expand
Subjective symptoms in idiopathic hypersomnia: beyond excessive sleepiness
TLDR
Mental fatigability, dependence on other people for awakening them, and a reduced benefit from usually alerting conditions (except being hyperactive or stressed) seem to be more specific of the daily problems of patients with idiopathic hypersomnia than daytime sleepiness. Expand
Electroencephalographic Markers of Idiopathic Hypersomnia: Where We are and Where We are Going
TLDR
Current and potential markers of idiopathic hypersomnia are presented, with a focus on sleep propensity, and EEG changes occurring in sleep deprivation conditions and recovery nights, as well as those observed in other central hypersomnolence conditions, namely narcolepsy type-1 and type-2. Expand
A comparison of idiopathic hypersomnia and narcolepsy-cataplexy using self report measures and sleep diary data.
  • D. Bruck, J. Parkes
  • Psychology, Medicine
  • Journal of neurology, neurosurgery, and psychiatry
  • 1996
TLDR
The IH group reported more consolidated nocturnal sleep, a lower propensity to nap, greater refreshment after naps, and a greater improvement in excessive daytime sleepiness since onset than the NLS group. Expand
Hypersomnia
TLDR
The causes of hypersomnia are detailed following the conventional classification of hypersomnic syndromes: narcolepsy, idiopathic hypersomnia, recurrent hypers insomnia, insufficient sleep syndrome, medication- and toxin-dependent sleepiness, hypersomnia associated with psychiatric disorders, hypers insomnia associated with neurological disorders, posttraumatic hypersomnia. Expand
Narcolepsy and Other Central Disorders of Hypersomnolence
TLDR
Hypnagogic hallucinations are realistic dream‐like experiences around sleep onset whereas the term 'hypnopompic' is used if they occur from awakening, and Pharmacotherapy for sleepiness should be tailored towards the individual patient. Expand
Polysomnographic study of nocturnal sleep in idiopathic hypersomnia without long sleep time
TLDR
A peculiar profile of microstructural sleep abnormalities may contribute to sleep fragmentation and, possibly, EDS in IH. Expand
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References

SHOWING 1-4 OF 4 REFERENCES
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TLDR
Polysomnographic recordings showed a spontaneous sleep period of 19.4 h and a normal Multiple Sleep Latency Test, which is clearly abnormal but essentially different from those of narcolepsy. Expand
Cataplexy and monoamine oxidase deficiency in Norrie disease
TLDR
The coexistence of cataplexy and abnormal REM sleep organization with Norrie disease is reported and it is suggested that abnormal MA0 activity or an imbalance between serotonin and other neurotransmitter levels may be involved in the pathogenesis of human catapLexy. Expand
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TLDR
A new syndrome called "delayed sleep phase insomnia" is described, which is proposed to be a disorder of the circadian sleep-wake rhythm in which the "advance" portion of the phase response curve is small. Expand
SLEEP AS A PROBLEM OF LOCALIZATION