IP 3-Dependent Ca 2+ Oscillations Switch into a Dual Oscillator Mechanism in the Presence of PLC-Linked Hormones

@article{Bartlett2019IP3C,
  title={IP 3-Dependent Ca 2+ Oscillations Switch into a Dual Oscillator Mechanism in the Presence of PLC-Linked Hormones},
  author={Paula J. Bartlett and Ielyaas Cloete and James Sneyd and Andrew P Thomas},
  journal={SSRN Electronic Journal},
  year={2019}
}

Short-term high fat diet feeding of mice suppresses catecholamine-stimulated Ca2+ signalling in hepatocytes and intact liver

It is proposed that impaired Ca2+ signalling plays a key role in the earliest phases of the etiology of NAFLD, and is responsible for many of the ensuing metabolic and related dysfunctional outcomes at the cellular and whole tissue level.

Purinergic P2Y1 and P2Y2/4 receptors elicit distinct Ca2+ signaling patterns in hepatocytes via differential feedback regulation by Protein Kinase C

Extracellular nucleotides are key regulators of liver physiology. In primary rat hepatocytes, P2Y1 receptor (P2Y1R) activation by ADP generates cytosolic calcium ([Ca2+]c) oscillations with narrow

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