Hypoxic augmentation of neuronal nicotinic acetylcholine receptors and carotid body function.

@article{Shirahata2003HypoxicAO,
  title={Hypoxic augmentation of neuronal nicotinic acetylcholine receptors and carotid body function.},
  author={Machiko Shirahata and Tomoko Higashi and Jeffrey A Mendoza and Serabi Hirasawa},
  journal={Advances in experimental medicine and biology},
  year={2003},
  volume={536},
  pages={
          269-75
        }
}
It is generally accepted that neurotransmitters are involved in hypoxic chemotransmission of the carotid body. The release of neurotransmitters from the glomus cell, a putative chemoreceptor cell, appears to be triggered by an influx of calcium and subsequent increase in intracellular calcium ([Ca2+]i). Several reports indicate that L-type and some other types of voltage-gated calcium channels are responsible for neurotranmitter release from glomus cells. Since these channels are activated by… CONTINUE READING
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