Hypothalamic-Pituitary-Adrenal Hypofunction in Myalgic Encephalomyelitis (ME)/Chronic Fatigue Syndrome (CFS) as a Consequence of Activated Immune-Inflammatory and Oxidative and Nitrosative Pathways

@article{Morris2016HypothalamicPituitaryAdrenalHI,
  title={Hypothalamic-Pituitary-Adrenal Hypofunction in Myalgic Encephalomyelitis (ME)/Chronic Fatigue Syndrome (CFS) as a Consequence of Activated Immune-Inflammatory and Oxidative and Nitrosative Pathways},
  author={G. Morris and G. Anderson and Michael Maes},
  journal={Molecular Neurobiology},
  year={2016},
  volume={54},
  pages={6806-6819}
}
  • G. Morris, G. Anderson, Michael Maes
  • Published 2016
  • Medicine, Biology
  • Molecular Neurobiology
  • There is evidence that immune-inflammatory and oxidative and nitrosative stress (O&NS) pathways play a role in the pathophysiology of myalgic encephalomyelitis (ME)/chronic fatigue syndrome (CFS). There is also evidence that these neuroimmune diseases are accompanied by hypothalamic-pituitary-adrenal (HPA) axis hypoactivity as indicated by lowered baseline glucocorticoid levels. This paper aims to review the bidirectional communications between immune-inflammatory and O&NS pathways and HPA axis… CONTINUE READING
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    References

    Publications referenced by this paper.
    SHOWING 1-10 OF 181 REFERENCES
    Hypothalamic-Pituitary-Adrenal Axis Function in Chronic Fatigue Syndrome
    • 108
    • PDF
    Glucocorticoids, cytokines and brain abnormalities in depression
    • 364
    The HPA axis and the genesis of chronic fatigue syndrome
    • 182
    • Highly Influential
    The neuroendocrinology of chronic fatigue syndrome.
    • 290
    • PDF
    Hypothalamic–pituitary–adrenal axis dysfunction in chronic fatigue syndrome
    • 181