Hypo-osmotic Shock-Induced Subclinical Inflammation of Skin in a Rat Model of Disrupted Skin Barrier Function

@article{Kishi2015HypoosmoticSS,
  title={Hypo-osmotic Shock-Induced Subclinical Inflammation of Skin in a Rat Model of Disrupted Skin Barrier Function},
  author={Chihiro Kishi and T. Minematsu and Lijuan Huang and Y. Mugita and A. Kitamura and G. Nakagami and Takumi Yamane and Mikako Yoshida and H. Noguchi and M. Funakubo and Taketoshi Mori and H. Sanada},
  journal={Biological Research For Nursing},
  year={2015},
  volume={17},
  pages={135 - 141}
}
Aging disrupts skin barrier function and induces xerosis accompanied by pruritus. In many cases, elderly patients complain of pruritus during skin hygiene care, a condition called aquagenic pruritus of the elderly (APE). To date, the pathophysiology and mechanism of action of APE have not been elucidated. We conducted the present study to test the hypothesis that hypo-osmotic shock of epidermal cells induces skin inflammation and elongation of C-fibers by nerve growth factor β (NGFβ) as a basic… Expand
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