Human rpL3 induces G₁/S arrest or apoptosis by modulating p21 (waf1/cip1) levels in a p53-independent manner.

@article{Russo2013HumanRI,
  title={Human rpL3 induces G₁/S arrest or apoptosis by modulating p21 (waf1/cip1) levels in a p53-independent manner.},
  author={Annapina Russo and Davide Esposito and Morena Catillo and Concetta Pietropaolo and Elvira Crescenzi and Giulia Russo},
  journal={Cell cycle},
  year={2013},
  volume={12 1},
  pages={76-87}
}
It is now largely accepted that ribosomal proteins may be implicated in a variety of biological functions besides that of components of the translation machinery. Many evidences show that a subset of ribosomal proteins are involved in the regulation of the cell cycle and apoptosis through modulation of p53 activity. In addition, p53-independent mechanisms of cell cycle arrest in response to alterations of ribosomal proteins availability have been described. Here, we identify human rpL3 as a new… CONTINUE READING

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