Human neutrophils switch to an activated phenotype after homing to the lung irrespective of inflammatory disease.

Abstract

Systemic inflammation can be investigated by changes in expression profiles of neutrophil receptors. Application of this technology for analysis of neutrophil phenotypes in diseased tissues is hampered by the absence of information regarding the modulation of neutrophil phenotypes after extravasation to tissues under non-inflammatory conditions. To fill… (More)
DOI: 10.1111/j.1365-2249.2008.03791.x

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