Homozygosity for angiotensinogen 235T variant increases the risk of myocardial infarction in patients with multi-vessel coronary artery disease.

@article{Olivieri2001HomozygosityFA,
  title={Homozygosity for angiotensinogen 235T variant increases the risk of myocardial infarction in patients with multi-vessel coronary artery disease.},
  author={Oliviero Olivieri and Chiara Stranieri and Domenico Girelli and Francesca Pizzolo and Silvia Grazioli and Carla Russo and Pier Franco Pignatti and Roberto Corrocher},
  journal={Journal of hypertension},
  year={2001},
  volume={19 5},
  pages={879-84}
}
OBJECTIVE Molecular variants of the angiotensinogen (AGT) and the angiotensin II type 1 receptor (ATR) genes have been associated with the risk of coronary artery disease (CAD) and myocardial infarction (MI), but data so far available are conflicting. The primary object of the paper is to verify this possible association by a rigorous, angiographically controlled study in a large sample of patients with or without multi-vessel CAD. DESIGN We designed a large case-control study in Italian… CONTINUE READING

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Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
Age , gender , smoking habit and number of stenosed vessels were the only differences between patients with or without previous myocardial infarction , who were similarly exposed to the other conventional risk factors ( including hypertension ) .
Age , gender , smoking habit and number of stenosed vessels were the only differences between patients with or without previous myocardial infarction , who were similarly exposed to the other conventional risk factors ( including hypertension ) .
AGT 235 T homozygous patients with multivessel CAD have an increased risk of myocardial infarction as compared with subjects with clinically similar phenotype but different genotype .
Homozygosity for angiotensinogen 235 T variant increases the risk of myocardial infarction in patients with multi - vessel coronary artery disease .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
AGT 235 T homozygous patients with multivessel CAD have an increased risk of myocardial infarction as compared with subjects with clinically similar phenotype but different genotype .
Homozygosity for angiotensinogen 235 T variant increases the risk of myocardial infarction in patients with multi - vessel coronary artery disease .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
AGT 235 T homozygous patients with multivessel CAD have an increased risk of myocardial infarction as compared with subjects with clinically similar phenotype but different genotype .
Homozygosity for angiotensinogen 235 T variant increases the risk of myocardial infarction in patients with multi - vessel coronary artery disease .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
AGT 235 T homozygous patients with multivessel CAD have an increased risk of myocardial infarction as compared with subjects with clinically similar phenotype but different genotype .
Homozygosity for angiotensinogen 235 T variant increases the risk of myocardial infarction in patients with multi - vessel coronary artery disease .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
AGT 235 T homozygous patients with multivessel CAD have an increased risk of myocardial infarction as compared with subjects with clinically similar phenotype but different genotype .
Homozygosity for angiotensinogen 235 T variant increases the risk of myocardial infarction in patients with multi - vessel coronary artery disease .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
AGT 235 T homozygous patients with multivessel CAD have an increased risk of myocardial infarction as compared with subjects with clinically similar phenotype but different genotype .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
Homozygosity for angiotensinogen 235 T variant increases the risk of myocardial infarction in patients with multi - vessel coronary artery disease .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
AGT 235 T homozygous patients with multivessel CAD have an increased risk of myocardial infarction as compared with subjects with clinically similar phenotype but different genotype .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
Homozygosity for angiotensinogen 235 T variant increases the risk of myocardial infarction in patients with multi - vessel coronary artery disease .
AGT 235 T homozygous patients with multivessel CAD have an increased risk of myocardial infarction as compared with subjects with clinically similar phenotype but different genotype .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
Homozygosity for angiotensinogen 235 T variant increases the risk of myocardial infarction in patients with multi - vessel coronary artery disease .
AGT 235 T homozygous patients with multivessel CAD have an increased risk of myocardial infarction as compared with subjects with clinically similar phenotype but different genotype .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
Homozygosity for angiotensinogen 235 T variant increases the risk of myocardial infarction in patients with multi - vessel coronary artery disease .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
Molecular variants of the angiotensinogen ( AGT ) and the angiotensin II type 1 receptor ( ATR ) genes have been associated with the risk of coronary artery disease ( CAD ) and myocardial infarction ( MI ) , but data so far available are conflicting .
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