Homocysteine and Thrombotic Disease

@inproceedings{DAngelo1997HomocysteineAT,
  title={Homocysteine and Thrombotic Disease},
  author={Armando D’Angelo},
  year={1997}
}
amino acid whose metabolism is at the intersection of The more severe cases of hyperhomocysteinemia are two metabolic pathways: remethylation and transsulfuration caused by homozygous defects in genes encoding for en(Fig 1). In remethylation, homocysteine acquires a methyl zymes of homocysteine metabolism. In such cases, a defect group from N-5-methyltetrahydrofolate (MTHF) or from bein an enzyme involved in either homocysteine remethylation taine to form methionine. The reaction with MTHF… CONTINUE READING
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