High-molecular-weight kininogen cleavage correlates with disease states in the bradykinin-mediated angioedema due to hereditary C1-inhibitor deficiency.

@article{Suffritti2014HighmolecularweightKC,
  title={High-molecular-weight kininogen cleavage correlates with disease states in the bradykinin-mediated angioedema due to hereditary C1-inhibitor deficiency.},
  author={C Suffritti and Andrea Zanichelli and Lorena Maggioni and Erika Bonanni and Massimo Cugno and Marco Cicardi},
  journal={Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology},
  year={2014},
  volume={44 12},
  pages={1503-14}
}
BACKGROUND The inherited deficiency of C1-inhibitor (C1-INH), which can be quantitative (type I) or qualitative (type II), is characterized by recurrent attacks of oedema, and it is known as hereditary angioedema due to C1-INH deficiency (HAE-C1-INH). The frequency of symptoms varies widely among patients and in the same patient during life. OBJECTIVE To identify laboratory markers of disease severity in HAE-C1-INH patients. METHODS We studied 162 patients with differently severe HAE-C1-INH… CONTINUE READING
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