Herpesvirus latency confers symbiotic protection from bacterial infection

@article{Barton2007HerpesvirusLC,
  title={Herpesvirus latency confers symbiotic protection from bacterial infection},
  author={Erik S. Barton and Douglas W White and Jason S. Cathelyn and Kelly A. Brett-McClellan and Michael Engle and Michael Steven Diamond and Virginia L. Miller and Herbert W. Virgin},
  journal={Nature},
  year={2007},
  volume={447},
  pages={326-329}
}
All humans become infected with multiple herpesviruses during childhood. After clearance of acute infection, herpesviruses enter a dormant state known as latency. Latency persists for the life of the host and is presumed to be parasitic, as it leaves the individual at risk for subsequent viral reactivation and disease. Here we show that herpesvirus latency also confers a surprising benefit to the host. Mice latently infected with either murine gammaherpesvirus 68 or murine cytomegalovirus… 
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TLDR
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