Hepcidin-25 in Chronic Hemodialysis Patients Is Related to Residual Kidney Function and Not to Treatment with Erythropoiesis Stimulating Agents

@inproceedings{Weerd2012Hepcidin25IC,
  title={Hepcidin-25 in Chronic Hemodialysis Patients Is Related to Residual Kidney Function and Not to Treatment with Erythropoiesis Stimulating Agents},
  author={Neelke C. van der Weerd and Muriel P. C. Grooteman and Michiel L. Bots and Marinus A. van den Dorpel and Claire H. den Hoedt and Albert H. A. Mazairac and Menso J. Nub{\'e} and Erik Lars Penne and Carlo A. J. M. Gaillard and Jack F. M. Wetzels and Erwin T G Wiegerinck and Dorine W Swinkels and Peter J Blankestijn and Piet Marten ter Wee},
  booktitle={PloS one},
  year={2012}
}
Hepcidin-25, the bioactive form of hepcidin, is a key regulator of iron homeostasis as it induces internalization and degradation of ferroportin, a cellular iron exporter on enterocytes, macrophages and hepatocytes. Hepcidin levels are increased in chronic hemodialysis (HD) patients, but as of yet, limited information on factors associated with hepcidin-25 in these patients is available. In the current cross-sectional study, potential patient-, laboratory- and treatment-related determinants of… CONTINUE READING

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