Glucose-dependent insulinotropic polypeptide (GIP) receptor deletion leads to reduced bone strength and quality.

@article{Mieczkowska2013GlucosedependentIP,
  title={Glucose-dependent insulinotropic polypeptide (GIP) receptor deletion leads to reduced bone strength and quality.},
  author={Aleksandra Mieczkowska and Nigel Irwin and Peter Raymond Flatt and Daniel Chappard and Guillaume Mabilleau},
  journal={Bone},
  year={2013},
  volume={56 2},
  pages={337-42}
}
Bone is permanently remodeled by a complex network of local, hormonal and neuronal factors that affect osteoclast and osteoblast biology. In this context, a role for gastro-intestinal hormones has been proposed based on evidence that bone resorption dramatically falls after a meal. Glucose-dependent insulinotropic polypeptide (GIP) is one of the candidate hormones as its receptor, glucose-dependent insulinotropic polypeptide receptor (GIPR), is expressed in bone. In the present study we… CONTINUE READING
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Glucose-dependent insulinotropic polypeptide receptor deficiency leads to modifications of trabecualr bone mass and quality in mice

  • C Gaudin-Audrain, N Irwin, S Mansur, PR Flatt, B Thorens, M Baslé
  • Bone
  • 2013
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Deficiency in glucose-dependent insulinotropic peptide receptor results in higher bone mass in male mice

  • G Mabilleau, C Gaudin-Audrain, N Irwin, PR Flatt, MF Basle, D. Chappard
  • Osteoporos Int 2012;23:S407–8
  • 2012

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