Glucoprivation increases estrogen receptor alpha immunoreactivity in the brain catecholaminergic neurons in ovariectomized rats.

@article{Reyes2001GlucoprivationIE,
  title={Glucoprivation increases estrogen receptor alpha immunoreactivity in the brain catecholaminergic neurons in ovariectomized rats.},
  author={Beverly A. S. Reyes and Maria Amelita C Estacio and Helen I'anson and Hiroko Tsukamura and Kenkou Maeda},
  journal={Neuroscience letters},
  year={2001},
  volume={299 1-2},
  pages={109-12}
}
Estrogen-dependent enhancement of glucoprivic-induced luteinizing hormone (LH) suppression is hypothesized to be due to increased estrogen receptor alpha (ERalpha)-immunoreactive (ir) cells in specific brain nuclei in a manner similar to fasting. ERalpha expression in various brain areas was determined in ovariectomized rats after systemic 2-deoxy-D-glucose (2DG)-induced glucoprivation. Expression of ERalpha in catecholaminergic neurons in the lower brainstem was also examined. ERalpha-ir cells… CONTINUE READING