Flicker sensitivity in treated ocular hypertension.

@article{Tytla1990FlickerSI,
  title={Flicker sensitivity in treated ocular hypertension.},
  author={M E Tytla and Graham E. Trope and J. Raymond Buncic},
  journal={Ophthalmology},
  year={1990},
  volume={97 1},
  pages={36-43}
}
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage, but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity (50-90%) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop (5-30%). The authors examined the possibility that the flicker losses in some of these patients represent not early glaucomatous damage, but instead a… CONTINUE READING

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Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
The ocular hypertensive patients were divided into three groups , which may reflect differing risks of glaucoma conversion .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
The ocular hypertensive patients were divided into three groups , which may reflect differing risks of glaucoma conversion .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Reductions in flicker sensitivity in ocular hypertension are thought to precede manifest glaucomatous damage , but the proportion of patients with ocular hypertension reported to have losses in flicker sensitivity ( 50 - 90% ) is far out of step with the proportion of ocular hypertensive patients in whom clinically defined glaucoma will develop ( 5 - 30% ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
Temporal contrast sensitivity was measured in 26 patients with ocular hypertension and in 22 patients with primary open - angle glaucoma ( POAG ) before and after hypotensive treatment ( timolol ) .
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