Flavivirus Infection Uncouples Translation Suppression from Cellular Stress Responses

Abstract

As obligate parasites, viruses strictly depend on host cell translation for the production of new progeny, yet infected cells also synthesize antiviral proteins to limit virus infection. Modulation of host cell translation therefore represents a frequent strategy by which viruses optimize their replication and spread. Here we sought to define how host cell… (More)
DOI: 10.1128/mBio.02150-16

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Cite this paper

@inproceedings{Roth2017FlavivirusIU, title={Flavivirus Infection Uncouples Translation Suppression from Cellular Stress Responses}, author={Hanna Roth and Vera Magg and Fabian Uch and Pascal Mutz and Philipp Klein and Katharina Haneke and Volker Lohmann and Ralf Bartenschlager and Oliver T. Fackler and Nicolas Locker and Georg Stoecklin and Alessia Ruggieri}, booktitle={mBio}, year={2017} }