Fear memories require protein synthesis in the amygdala for reconsolidation after retrieval

@article{Nader2000FearMR,
  title={Fear memories require protein synthesis in the amygdala for reconsolidation after retrieval},
  author={Karim Nader and Glenn E. Schafe and Joseph E. Le Doux},
  journal={Nature},
  year={2000},
  volume={406},
  pages={722-726}
}
‘New’ memories are initially labile and sensitive to disruption before being consolidated into stable long-term memories. Much evidence indicates that this consolidation involves the synthesis of new proteins in neurons. The lateral and basal nuclei of the amygdala (LBA) are believed to be a site of memory storage in fear learning. Infusion of the protein synthesis inhibitor anisomycin into the LBA shortly after training prevents consolidation of fear memories. Here we show that consolidated… 
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TLDR
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Reconolidation can be shown to exist in social memory, and some of its boundary conditions are discussed.
Glucocorticoid receptors in the basolateral nucleus of amygdala are required for postreactivation reconsolidation of auditory fear memory
TLDR
There is strong evidence that glucocorticoid receptors in the basolateral nucleus of amygdala (BLA) are required for reconsolidation as well as consolidation of AFM.
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