Corpus ID: 208815408

Expression of vascular endothelial growth factor and glial fibrillary acidic protein in a rat model of traumatic brain injury treated with honokiol: a biochemical and immunohistochemical study

@inproceedings{Cetin2019ExpressionOV,
  title={Expression of vascular endothelial growth factor and glial fibrillary acidic protein in a rat model of traumatic brain injury treated with honokiol: a biochemical and immunohistochemical study},
  author={Abdurrahman Cetin and Engin Deveci},
  year={2019}
}
684 Address for correspondence: Prof. E. Deveci, PhD, Dicle University, Faculty of Medicine, Department of Histology and Embryology, 21280, Diyarbakır, Turkey, tel: +90 412 2488001 ext. 4443 (faculty room), fax: +90 412 2488440, e-mail: devecie32@hotmail.com Expression of vascular endothelial growth factor and glial fibrillary acidic protein in a rat model of traumatic brain injury treated with honokiol: a biochemical and immunohistochemical study 

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The results suggest that pericyte involvement in adaptive angiogenesis may be altered following TBI, and show a distinct expression pattern of angiogenic factors following injury. Expand
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Investigation of the alteration of glial fibrillary acidic protein (GFAP) immunoreactivity in developing rat brain after intrauterine infection induces an increased expression of GFAP in the neonatal brain. Expand
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The results of measuring the concentrations of both GFAP and S-100 protein in the blood of patients with acute severe head trauma and healthy controls are described, using the dissociation-enhanced lanthanide fluorescence immunoassay (DELFIA) system for detection of GFAP. Expand
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Honokiol has a potent anti-inflammatory effect in cerebral ischemia-reperfusion mice and this effect might be attributed to its inhibition ability on the NF-κB activation, consequently blocking the production of inflammatory factors including: NO, tumor necrosis factor-α (TNF-α) and RANTES/CCL5 in glial cells. Expand
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