Expression of myeloid differentiation factor 88 in neurons is not requisite for the induction of sickness behavior by interleukin-1β

@inproceedings{Braun2012ExpressionOM,
  title={Expression of myeloid differentiation factor 88 in neurons is not requisite for the induction of sickness behavior by interleukin-1β},
  author={Theodore P. Braun and Aaron J Grossberg and Biliana O. Veleva-Rotse and Julia E. Maxson and Marek Szumowski and Anthony P. Barnes and Daniel L Marks},
  booktitle={Journal of Neuroinflammation},
  year={2012}
}
Animals respond to inflammation by suppressing normal high-energy activities, including feeding and locomotion, in favor of diverting resources to the immune response. The cytokine interleukin-1 beta (IL-1β) inhibits normal feeding and locomotor activity (LMA) via its actions in the central nervous system (CNS). Behavioral changes in response to IL-1β are mediated by myeloid differentiation factor 88 (MyD88) in non-hematopoietic cells. It is unknown whether IL-1β acts directly on neurons or… CONTINUE READING
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