The relationship between oxidative stress and exercise
- BiologyJournal of basic and clinical physiology and pharmacology
Possible partial answers are given, all of which lend strong support to the conclusion that the network responsible for homeostasis of the redox status is very effective, however, much more data are required to address the association between exercise and OS and its dependence on various relevant factors.
Acute exercise and oxidative stress: a 30 year history
- BiologyDynamic medicine : DM
A comprehensive summary of original investigations focused on exercise-induced oxidative stress is presented to provide the reader with a well-documented account of the research done within this area of science over the past 30 years.
Polyphenols in Exercise Performance and Prevention of Exercise-Induced Muscle Damage
- BiologyOxidative medicine and cellular longevity
This review focuses on those polyphenols, present in the plant kingdom, that have been recently suggested to exert some positive effects on exercise-induced muscle damage and oxidative stress.
The effect of exercise on oxidative stress and other health markers: exploring new technology and methodology
The finding from this thesis suggests that brain glutathione of young sedentary men as measured by magnetic resonance spectroscopy was altered in response to acute exercise, in an exercise intensity dependent manner.
Physical exercise and catecholamines response: benefits and health risk: possible mechanisms
- BiologyFree radical research
Results show a significant role of CATs in carbohydrate and lipid metabolism, immunity and as generators of ROS, depending on PE intensity and duration, and mechanisms of cytotoxic free radicals and reactive oxygen species (ROS) generation during CATs oxidation.
Oxidative Stress in Training, Overtraining and Detraining: from Experimental to Applied Research
According to the hormesis theory, the responses of biological systems to stressors in exercise training may be explained by a U-shaped curve with inactivity and overtraining as the two endpoints, and it seems reasonable to assume that changes in ADS are also reversible.
Exercise-induced oxidative stress: A tool for “hormesis” and “adaptive response”
- Biology, Psychology
A brief review provides an overview of several conceptual frameworks related to exercise-mediated hormetic adaptive responses rather than a detailed critique of individual reports, and indicates that exercise-related beneficial adaptations are strongly regulated by exercise-induced oxidative stress, consistent with hormesis theory.
Effect of Exercise on Oxidative Stress in Neurological Disorders
Regular moderate exercise improves cardiovascular and cerebrovascular fitness in normal older subjects and in patients with stroke, SCI, TBI, AD, PD, and depression by increasing blood flow and improves cognition and support learning and memory.
Hesperidin Functions as an Ergogenic Aid by Increasing Endothelial Function and Decreasing Exercise-Induced Oxidative Stress and Inflammation, Thereby Contributing to Improved Exercise Performance
The results collected in this review show that hesperidin improves endothelial function, inhibits ROS production, decreases production and plasma levels of pro-inflammatory markers, and improves anaerobic exercise outcomes (e.g., power, speed, energy).
SHOWING 1-10 OF 118 REFERENCES
Adaptation to exercise-induced oxidative stress: from muscle to brain.
- BiologyExercise immunology review
Regular exercise causes adaptation of the antioxidant and repair systems, which could result in a decreased base level of oxidative damage and increased resistance to oxidative stress.
Exercise and hormesis: oxidative stress-related adaptation for successful aging
It is suggested that exercise-induced ROS production plays a role in the induction of antioxidants, DNA repair and protein degrading enzymes, resulting in decreases in the incidence of oxidative stress-related diseases and retardation of the aging process.
The effect of exercise training on oxidative damage of lipids, proteins, and DNA in rat skeletal muscle: evidence for beneficial outcomes.
- BiologyFree radical biology & medicine
The effects of moderate, strenuous, and overtraining on oxidative stress markers and DNA repair in rat liver.
- BiologyCanadian journal of applied physiology = Revue canadienne de physiologie appliquee
The findings of this study imply that overtraining induces oxidative damage to nuclear DNA, but not to liver lipids and proteins, as well as lipid peroxidation and protein oxidation in exercising animals.
Exercise and the immune system: regulation, integration, and adaptation.
- Medicine, BiologyPhysiological reviews
Con considerations of the clinical ramifications of exercise in the prevention of diseases for which the immune system has a role is of importance, and the interactions between exercise and infectious diseases as well as exercise and neoplasia within the context of both aging and nutrition are addressed.
Exercise and Hormesis
- BiologyAnnals of the New York Academy of Sciences
It is suggested that ROS may serve as messenger molecules to activate adaptive responses through these redox‐sensitive signaling pathways to maintain cellular oxidant‐antioxidant homeostasis during exercise.
Exercise training-induced alterations in skeletal muscle antioxidant capacity: a brief review.
- BiologyMedicine and science in sports and exercise
The effects of chronic exercise on the up-regulation of both antioxidant enzymes and the glutathione antioxidant defense system are discussed and it seems plausible that increased cellular concentrations of these antioxidants will reduce the risk of cellular injury, improve performance, and delay muscle fatigue.
Energy intake, meal frequency, and health: a neurobiological perspective.
- Medicine, BiologyAnnual review of nutrition
A better understanding of the neurobiological mechanisms by which meal size and frequency affect human health may lead to novel approaches for disease prevention and treatment.
Oxidative stress in marathon runners: interest of antioxidant supplementation
- BiologyBritish Journal of Nutrition
It is found that exercise causes an increase in the activity of plasma xanthine oxidase and an activation of NF-κB in peripheral blood lymphocytes after marathon running and evidence in man is reported that reactive oxygen species act as signals in exercise as decreasing their formation prevents activation of important signalling pathways which can cause useful adaptations in cells.
Decreasing xanthine oxidase‐mediated oxidative stress prevents useful cellular adaptations to exercise in rats
- BiologyThe Journal of physiology
Evidence is reported that decreasing RONS formation prevents activation of important signalling pathways, predominantly the MAPK–NF‐κB pathway; consequently the practice of taking antioxidants before exercise may have to be re‐evaluated.