Evidence that ‘food addiction’ is a valid phenotype of obesity

  title={Evidence that ‘food addiction’ is a valid phenotype of obesity},
  author={Caroline A. Davis and Claire Curtis and Robert Levitan and Jacqueline C Carter and Allan S. Kaplan and James L. Kennedy},

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The co-occurrence of FA in treatment-seeking GD patients is related to poorer emotional and psychological states and treatment interventions and related behavioral addictions should consider potential associations with problematic eating behavior and aim to include techniques that aid patients in better managing this behavior.
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Available data suggest that FA seems to be a transnosografic construct and exists in all EDs, with higher prevalence in Bulimia Nervosa.
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This study provided evidence that the French–Canadian version of the AEBS is a valid measure of food addiction, but it did not permit to establish advantages over YFAS 2.0 with a clinical sample.
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Similar patterns of neural activation are implicated in addictive-like eating behavior and substance dependence: elevated activation in reward circuitry in response to food cues and reduced activation of inhibitory regions in responseto food intake.
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It is suggested that the inattentive and impulsive behaviors that characterize ADHD could contribute to overeating in the current food environment, with its emphasis on fast food consumption and its many food temptations.
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Work presented in this review strongly supports the notion that food addiction is a real phenomenon, and indicates that traditional pharmacological and behavioral interventions for other substance-use disorders may prove useful in treating obesity.
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Healthy, normal weight individuals, by definition, do not suffer from food addiction; however, overweight and obese individuals could meet clinical criteria.
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