Eroded Versus Ruptured Plaques at the Culprit Site of STEMI: In Vivo Pathophysiological Features and Response to Primary PCI.

@article{Saia2015ErodedVR,
  title={Eroded Versus Ruptured Plaques at the Culprit Site of STEMI: In Vivo Pathophysiological Features and Response to Primary PCI.},
  author={Francesco Saia and Kenichi Komukai and Davide Capodanno and Vasile Sirbu and Giuseppe Musumeci and Giacomo G Boccuzzi and Giuseppe Tarantini and Massimo Fineschi and Gabriele Tumminello and Chiara Bernelli and Giampaolo Niccoli and Micol Coccato and Barbara Bordoni and Hiram G. Bezerra and Giuseppe G. L. Biondi-Zoccai and Renu Virmani and Giulio Guagliumi},
  journal={JACC. Cardiovascular imaging},
  year={2015},
  volume={8 5},
  pages={566-575}
}
OBJECTIVES The aim of this study was to evaluate the pathophysiological features and response to primary percutaneous coronary intervention (PCI) of nonruptured/eroded plaque versus ruptured plaque as a cause of ST-segment elevation myocardial infarction (STEMI). BACKGROUND Autopsy series identified nonruptured/eroded plaque and ruptured plaque as the principal pathological substrates underlying coronary thrombosis in STEMI. The real incidence of different plaque morphologies, associated… CONTINUE READING
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