Enalapril treatment alters the contribution of epoxyeicosatrienoic acids but not gap junctions to endothelium-derived hyperpolarizing factor activity in mesenteric arteries of spontaneously hypertensive rats.

@article{Ellis2009EnalaprilTA,
  title={Enalapril treatment alters the contribution of epoxyeicosatrienoic acids but not gap junctions to endothelium-derived hyperpolarizing factor activity in mesenteric arteries of spontaneously hypertensive rats.},
  author={Anthie Ellis and Kenichi Goto and Daniel J. Chaston and Therese D. Brackenbury and Kate R Meaney and John R Falck and Richard J. H. Wojcikiewicz and Caryl E. Hill},
  journal={The Journal of pharmacology and experimental therapeutics},
  year={2009},
  volume={330 2},
  pages={
          413-22
        }
}
Reduction in endothelium-derived hyperpolarizing factor (EDHF)-mediated dilatory function in large, elastic arteries during hypertension is reversed after blood pressure normalization. We investigated whether similar mechanisms occurred in smaller mesenteric resistance arteries from aged Wistar-Kyoto (WKY) rats, spontaneously hypertensive rats (SHRs), and SHRs treated with the angiotensin-converting enzyme inhibitor, enalapril, using immunohistochemistry, serial-section electron microscopy… CONTINUE READING

Citations

Publications citing this paper.
SHOWING 1-10 OF 14 CITATIONS

The endothelium in compliance and resistance vessels.

  • Frontiers in bioscience
  • 2011
VIEW 7 EXCERPTS
CITES BACKGROUND
HIGHLY INFLUENCED

References

Publications referenced by this paper.
SHOWING 1-10 OF 40 REFERENCES

Angiotensin II receptor blockade corrects altered expression of gap junctions in vascular endothelial cells from hypertensive rats.

  • American journal of physiology. Heart and circulatory physiology
  • 2004
VIEW 5 EXCERPTS
HIGHLY INFLUENTIAL

Similar Papers