Effect of Marine Omega 3 Fatty Acids on Methylmercury-Induced Toxicity in Fish and Mammalian Cells In Vitro

@inproceedings{Nstbakken2012EffectOM,
  title={Effect of Marine Omega 3 Fatty Acids on Methylmercury-Induced Toxicity in Fish and Mammalian Cells In Vitro},
  author={Ole Jakob N\ostbakken and I. L. Bredal and P{\aa}l A Olsvik and Tien-Sheng Huang and Bente Elisabeth Torstensen},
  booktitle={Journal of biomedicine & biotechnology},
  year={2012}
}
Methylmercury (MeHg) is a ubiquitous environmental contaminant which bioaccumulates in marine biota. Fish constitute an important part of a balanced human diet contributing with health beneficial nutrients but may also contain contaminants such as MeHg. Interactions between the marine n-3 fatty acids eicosapentaenoic acid (20:5n-3, EPA) and docosahexaenoic acid (22:6n-3, DHA) with MeHg-induced toxicity were investigated. Different toxic and metabolic responses were studied in Atlantic salmon… CONTINUE READING

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Interactions between the marine n-3 fatty acids eicosapentaenoic acid ( 20:5n-3 , EPA ) and docosahexaenoic acid ( 22:6n-3 , DHA ) with MeHg - induced toxicity were investigated .
Interactions between the marine n-3 fatty acids eicosapentaenoic acid ( 20:5n-3 , EPA ) and docosahexaenoic acid ( 22:6n-3 , DHA ) with MeHg - induced toxicity were investigated .
Interactions between the marine n-3 fatty acids eicosapentaenoic acid ( 20:5n-3 , EPA ) and docosahexaenoic acid ( 22:6n-3 , DHA ) with MeHg - induced toxicity were investigated .
In conclusion , marine n-3 fatty acids may ameliorate MeHg toxicity , either by decreasing apoptosis ( EPA ) or by reducing MeHg uptake ( DHA ) .
Interactions between the marine n-3 fatty acids eicosapentaenoic acid ( 20:5n-3 , EPA ) and docosahexaenoic acid ( 22:6n-3 , DHA ) with MeHg - induced toxicity were investigated .
Interactions between the marine n-3 fatty acids eicosapentaenoic acid ( 20:5n-3 , EPA ) and docosahexaenoic acid ( 22:6n-3 , DHA ) with MeHg - induced toxicity were investigated .
Interactions between the marine n-3 fatty acids eicosapentaenoic acid ( 20:5n-3 , EPA ) and docosahexaenoic acid ( 22:6n-3 , DHA ) with MeHg - induced toxicity were investigated .
In conclusion , marine n-3 fatty acids may ameliorate MeHg toxicity , either by decreasing apoptosis ( EPA ) or by reducing MeHg uptake ( DHA ) .
Interactions between the marine n-3 fatty acids eicosapentaenoic acid ( 20:5n-3 , EPA ) and docosahexaenoic acid ( 22:6n-3 , DHA ) with MeHg - induced toxicity were investigated .
Interactions between the marine n-3 fatty acids eicosapentaenoic acid ( 20:5n-3 , EPA ) and docosahexaenoic acid ( 22:6n-3 , DHA ) with MeHg - induced toxicity were investigated .
Interactions between the marine n-3 fatty acids eicosapentaenoic acid ( 20:5n-3 , EPA ) and docosahexaenoic acid ( 22:6n-3 , DHA ) with MeHg - induced toxicity were investigated .
In conclusion , marine n-3 fatty acids may ameliorate MeHg toxicity , either by decreasing apoptosis ( EPA ) or by reducing MeHg uptake ( DHA ) .
Interactions between the marine n-3 fatty acids eicosapentaenoic acid ( 20:5n-3 , EPA ) and docosahexaenoic acid ( 22:6n-3 , DHA ) with MeHg - induced toxicity were investigated .
Interactions between the marine n-3 fatty acids eicosapentaenoic acid ( 20:5n-3 , EPA ) and docosahexaenoic acid ( 22:6n-3 , DHA ) with MeHg - induced toxicity were investigated .
In conclusion , marine n-3 fatty acids may ameliorate MeHg toxicity , either by decreasing apoptosis ( EPA ) or by reducing MeHg uptake ( DHA ) .
Interactions between the marine n-3 fatty acids eicosapentaenoic acid ( 20:5n-3 , EPA ) and docosahexaenoic acid ( 22:6n-3 , DHA ) with MeHg - induced toxicity were investigated .
Interactions between the marine n-3 fatty acids eicosapentaenoic acid ( 20:5n-3 , EPA ) and docosahexaenoic acid ( 22:6n-3 , DHA ) with MeHg - induced toxicity were investigated .
Interactions between the marine n-3 fatty acids eicosapentaenoic acid ( 20:5n-3 , EPA ) and docosahexaenoic acid ( 22:6n-3 , DHA ) with MeHg - induced toxicity were investigated .
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