Early pathophysiological alterations in experimental cardiomyopathy: the Syrian cardiomyopathic hamster.

@article{Escobales2008EarlyPA,
  title={Early pathophysiological alterations in experimental cardiomyopathy: the Syrian cardiomyopathic hamster.},
  author={Nelson Escobales and Maria J Crespo},
  journal={Puerto Rico health sciences journal},
  year={2008},
  volume={27 4},
  pages={307-14}
}
The Syrian cardiomyopathic hamster (SCH) is an established animal model for genetic cardiomyopathy. The disease in the hamster develops through similar stages to those observed in humans with this condition. The pathophysiological basis for this condition in the hamster resides in an inherited mutation in the gene encoding for delta-sarcoglycan, a component of the dystrophin complex. Two basic mechanisms contribute to cardiomyopathy in this model: ischemic heart disease by vasospasms of the… CONTINUE READING

Citations

Publications citing this paper.
Showing 1-7 of 7 extracted citations

References

Publications referenced by this paper.
Showing 1-10 of 79 references

Coronary microvascular dysfunction.

The New England journal of medicine • 2007

The role of bradykinin b 1 receptor on cardiac remodeling in strokeprone spontaneously hypertensive rats ( SHRSP )

N Moniwa, J Agatha, M Hagiwara, N Ura, K Shimamoto
biol Chem • 2007

Enhanced Contractility of Ang II in the Aorta of Cardiomyopathic Hamsters is Mediated by an Increased Ang IIbinding Capacity and Release of ET1

MJ Crespo, PI Altieri, N Escobales
Vasc Pharmacol • 2006

Similar Papers

Loading similar papers…