ECG changes in patients with acute ethanol intoxication

@article{Aaseb2007ECGCI,
  title={ECG changes in patients with acute ethanol intoxication},
  author={Willy Aaseb{\o} and Jan Erikssen and J{\o}rgen Jonsbu and Knut Stavem},
  journal={Scandinavian Cardiovascular Journal},
  year={2007},
  volume={41},
  pages={79 - 84}
}
Objectives. To assess how ethanol in potential lethal serum concentrations affects features of the ECG that may be associated with cardiac arrhythmias. Design. We included 84 patients, who were hospitalised with assumed acute ethanol intoxication. In the emergency room resting ECG was recorded and blood was collected for serum osmolality measurement used as a proxy for ethanol level. Thirty-two also had ECG recorded at discharge. Twenty-seven hospitalised patients without known alcohol… 

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References

SHOWING 1-10 OF 27 REFERENCES

Clinical features in poisonings by tricyclic antidepressants with special reference to the ECG.

Clinical variables, and especially their relation to the ECG, have been studied in 153 cases of poisonings by tricyclic antidepressants (TCA). The mean age of the patients was 34 years. Amitriptyline

Acute alcohol intake and P-wave dispersion in healthy men.

TLDR
Acute moderate dose of alcohol intake in short time is associated with an increase in Pmax and Pd, which are electrocardiographic markers used to evaluate the discontinuous propagation of sinus impulses and the prolongation of atrial conduction time, respectively.

Serum osmolality in acute intoxication: a prospective clinical study.

TLDR
The delta-Osm/ADH procedure provides an efficient, rapid, and readily available method to evaluate the acutely intoxicated patient for the presence of EtOH and/or other low Mr volatiles.

Alcohol, cardiac arrhythmias and sudden death.

TLDR
Several lines of evidence suggest that heavy drinking increases the risk of sudden cardiac death with fatal arrhythmia as the most likely mechanism and the effect of social drinking on clinical arrhythmias in non-alcoholic cardiac patients needs to be addressed.

Increased osmolal gap in alcoholic acidosis.

TLDR
Elevations of endogenous glycerol, acetone, and acetone metabolite levels should now be added as causes for an increased osmolal gap in the alcoholic patient.

Non-cardiac QTc-prolonging drugs and the risk of sudden cardiac death.

TLDR
The use of non-cardiac QTc-prolonging drugs in a general population is associated with an increased risk of sudden cardiac death.

Effects of acute alcohol infusion on duration and dispersion of QT interval in male patients with coronary artery disease and in healthy controls

TLDR
Whether acute alcohol intoxication prolongs repolarization in patients with stable coronary artery disease (CAD) is studied to find out if it increases the risk of sudden coronary death.