Dysregulation of neuronal calcium homeostasis in Alzheimer's disease - A therapeutic opportunity?

Abstract

Alzheimer's disease (AD) is the disease of lost memories. Synaptic loss is a major reason for memory defects in AD. Signaling pathways involved in memory loss in AD are under intense investigation. The role of deranged neuronal calcium (Ca2+) signaling in synaptic loss in AD is described in this review. Familial AD (FAD) mutations in presenilins are linked… (More)
DOI: 10.1016/j.bbrc.2016.09.053

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