Disruption of the vacuolar calcium-ATPases in Arabidopsis results in the activation of a salicylic acid-dependent programmed cell death pathway.

@article{Boursiac2010DisruptionOT,
  title={Disruption of the vacuolar calcium-ATPases in Arabidopsis results in the activation of a salicylic acid-dependent programmed cell death pathway.},
  author={Yann Boursiac and Sang Min Lee and Shawn M. Romanowsky and Robert R. Blank and Chris Sladek and Woo Sik Chung and Jeffrey F. Harper},
  journal={Plant physiology},
  year={2010},
  volume={154 3},
  pages={
          1158-71
        }
}
Calcium (Ca(2+)) signals regulate many aspects of plant development, including a programmed cell death pathway that protects plants from pathogens (hypersensitive response). Cytosolic Ca(2+) signals result from a combined action of Ca(2+) influx through channels and Ca(2+) efflux through pumps and cotransporters. Plants utilize calmodulin-activated Ca(2+) pumps (autoinhibited Ca(2+)-ATPase [ACA]) at the plasma membrane, endoplasmic reticulum, and vacuole. Here, we show that a double knockout… CONTINUE READING
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