Disruption of PAMP-induced MAP kinase cascade by a Pseudomonas syringae effector activates plant immunity mediated by the NB-LRR protein SUMM2.

@article{Zhang2012DisruptionOP,
  title={Disruption of PAMP-induced MAP kinase cascade by a Pseudomonas syringae effector activates plant immunity mediated by the NB-LRR protein SUMM2.},
  author={Zhibin Zhang and Yaling Wu and Minghui Gao and Jie Zhang and Qing Kong and Yanan Liu and Hongping Ba and Jianmin Zhou and Yuelin Zhang},
  journal={Cell host & microbe},
  year={2012},
  volume={11 3},
  pages={253-63}
}
Pathogen-associated molecular pattern (PAMP)-triggered immunity (PTI) serves as a primary plant defense response against microbial pathogens, with MEKK1, MKK1/MKK2, and MPK4 functioning as a MAP kinase cascade downstream of PAMP receptors. Plant Resistance (R) proteins sense specific pathogen effectors to initiate a second defense mechanism, termed effector-triggered immunity (ETI). In a screen for suppressors of the mkk1 mkk2 autoimmune phenotype, we identify the nucleotide-binding leucine… CONTINUE READING
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