Differential regulation of p38 mitogen-activated protein kinase mediates gender-dependent catecholamine-induced hypertrophy.

Rajesh C. Dash, Albrecht G. Schmidt, +6 authors Evangelia G Kranias
Published 2003 in Cardiovascular research

Abstract

OBJECTIVE Exogenous catecholamine exposure has been associated with p38 mitogen-activated protein kinase (MAPK) and cardiac hypertrophy. In this study, we investigated the regulation of p38 MAPK in cardiac remodeling elicited by endogenous adrenergic mechanisms. METHODS Transgenic male and female mice with fourfold phospholamban (PLB) overexpression… (More)

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Differential regulation of p38 mitogen-activated protein kinase mediates gender-dependent catecholamine-induced hypertrophy.

Abstract

Topics

6 Figures and Tables

Cited By

Effects of myostatin deletion in aging mice.

Gender modulates cardiac phenotype development in genetically modified mice.

A Systems Biology Approach to Investigating Sex Differences in Cardiac Hypertrophy

Blood flow-restricted walking in older women: does the acute hormonal response associate with muscle hypertrophy?

Gender differences in cardiac hypertrophic remodeling.

Mitogen-activated protein kinase phosphatase-1: a critical phosphatase manipulating mitogen-activated protein kinase signaling in cardiovascular disease (review).

Nuclear pore rearrangements and nuclear trafficking in cardiomyocytes from rat and human failing hearts

N-acetylcysteine reverses cardiac myocyte dysfunction in a rodent model of behavioral stress.

Ablation of cyclase-associated protein 2 (CAP2) leads to cardiomyopathy

Paradoxical impairment of angiogenesis, endothelial function and circulating number of endothelial progenitor cells in DPP4-deficient rat after critical limb ischemia

References

Cardiac-specific oververting enzyme inhibition

17beta-estradiol attenuates the development of pressure-overload hypertrophy.

Interactions between 2000;141:4629–4636. phospholamban and beta-adrenergic drive may lead to car

Dual specificity phosphatases: a gene family for control of MAP kinase function.

Differential effects of 1999;147:1129–1136

Important role of Biol Chem 2002;277:9387–9394. endogenous norepinephrine and epinephrine in the development

p38 Mitogen-activated protein overload

Conditional expression of the mitogen

The dual-specificity report

A Role for the p38 Mitogen-activated Protein Kinase Pathway in Myocardial Cell Growth, Sarcomeric Organization, and Cardiac-specific Gene Expression

Slides referencing similar topics

Differential regulation of p38 mitogen-activated protein kinase mediates gender-dependent catecholamine-induced hypertrophy.

Abstract

Topics

6 Figures and Tables

Cited By

Effects of myostatin deletion in aging mice.

Gender modulates cardiac phenotype development in genetically modified mice.

A Systems Biology Approach to Investigating Sex Differences in Cardiac Hypertrophy

Blood flow-restricted walking in older women: does the acute hormonal response associate with muscle hypertrophy?

Gender differences in cardiac hypertrophic remodeling.

Mitogen-activated protein kinase phosphatase-1: a critical phosphatase manipulating mitogen-activated protein kinase signaling in cardiovascular disease (review).

Nuclear pore rearrangements and nuclear trafficking in cardiomyocytes from rat and human failing hearts

N-acetylcysteine reverses cardiac myocyte dysfunction in a rodent model of behavioral stress.

Ablation of cyclase-associated protein 2 (CAP2) leads to cardiomyopathy

Paradoxical impairment of angiogenesis, endothelial function and circulating number of endothelial progenitor cells in DPP4-deficient rat after critical limb ischemia

References

Cardiac-specific oververting enzyme inhibition

17beta-estradiol attenuates the development of pressure-overload hypertrophy.

Interactions between 2000;141:4629–4636. phospholamban and beta-adrenergic drive may lead to car

Dual specificity phosphatases: a gene family for control of MAP kinase function.

Differential effects of 1999;147:1129–1136

Important role of Biol Chem 2002;277:9387–9394. endogenous norepinephrine and epinephrine in the development

p38 Mitogen-activated protein overload

Conditional expression of the mitogen

The dual-specificity report

A Role for the p38 Mitogen-activated Protein Kinase Pathway in Myocardial Cell Growth, Sarcomeric Organization, and Cardiac-specific Gene Expression

Similar Papers

Slides referencing similar topics