Developing teeth as biomarker of dioxin exposure

@article{Alaluusua1999DevelopingTA,
  title={Developing teeth as biomarker of dioxin exposure},
  author={Satu Alaluusua and P. L. Lukinmaa and Jorma Torppa and Jouko Tuomisto and Terttu Vartiainen},
  journal={The Lancet},
  year={1999},
  volume={353}
}

Bone toxicity of persistent organic pollutants

TLDR
Skeletal properties of rats exposed maternally to 2,3,7,8tetrachlorodibenzo-p-dioxin, Northern Contaminant Mixture and Aroclor 1254 indicate that POPs decrease bone strength, but the interpretation is difficult as there is more trabecular bone within cortical bone with compromised quality and increased porosity.

EFFECTS OF DIOXINS AND PCBS ON THE HUMAN ORGANISM AND THEIR INFLUENCE ON DEVELOPING TEETH OF CHILDREN VIA MOTHER

  • 2011
Polychlórované dibenzo-p-dioxíny (PCDD), dibenzofurány (PCDF) a bifenyly (PCB) tvoria skupinu stabilných toxických chemických látok, ktoré ovplyvňujú ľudské zdravie a životné prostredie. Tieto

7,12-dimethylbenz[a]anthracene interferes with the development of cultured mouse mandibular molars.

TLDR
The toxic influence of PAHs on tooth development in vitro shows enamel formation more severely than dentin formation and occasionally prevented amelogenesis completely, in line with the finding that maternal smoking during pregnancy has an adverse effect on child's tooth development.

The Effects of TCDD on the Development of Teeth and Cortical Bone in Rats: Implications for Risk Assessment

TLDR
The present study describes new developmental defects induced by TCDD: alterations in tooth and bone development using three differentially sensitive rat lines and demonstrated that the development of rodent dentition is disturbed at very low doses that are relevant for risk assessment.

Toxicity of dioxin to developing teeth and salivary glands : An experimental study

TLDR
This document summarizes current capabilities, research and operational priorities, and plans for further studies that were established at the 2015 USGS workshop on quantitative hazard assessments of earthquake-triggered landsliding and liquefaction in the Central American region.

Developmental Dental Aberrations After the Dioxin Accident in Seveso

TLDR
In conclusion, developmental dental aberrations were associated with childhood exposure to TCDD, and the hypothesis that dioxins can interfere with human organogenesis was supported.

Molaren-Inzisiven-Hypomineralisation (MIH)

Die Molaren-Inzisiven-Hypomineralisation (MIH) – auch bekannt unter dem Synonym der Kreidezähne – stellt heute neben der Karies die zweithäufigste Erkrankung an Zähnen im Kindes- und Jugendalter dar.

Association Study on Nutrition in the First Year of Life and Molar-Incisor Hypomineralization (MIH)—Results from the GINIplus and LISA Birth Cohort Studies

TLDR
The results of this study suggest the lack of an association between early nutrition in the first year of life and MIH in the permanent dentition.

Molaren-Inzisiven-Hypomineralisation (MIH). Häufigkeit und mögliche Ursachen unter besonderer Berücksichtigung der Ergebnisse aus den Münchner Geburtskohorten GINIplus und LISA

Die Molaren-Inzisiven-Hypomineralisation (MIH) – mittlerweile auch bekannt unter dem Begriff der Kreidezähne – stellt heute neben der Karies eine häufige Erkrankung der Zähne im Kindes- und
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References

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Epidermal growth factor receptor as a mediator of developmental toxicity of dioxin in mouse embryonic teeth.

TLDR
Results show that the interference of TCDD with mouse molar tooth development in vitro involves EGFR signaling, which means that EGFR may also play a role in the developmental defects that dioxins cause in human teeth.