Denture cream

@article{Nations2008DentureC,
  title={Denture cream},
  author={S. Nations and P. Boyer and L. Love and M. Burritt and J. Butz and G. Wolfe and L. Hynan and J. Reisch and J. Trivedi},
  journal={Neurology},
  year={2008},
  volume={71},
  pages={639 - 643}
}
Background: Chronic, excess zinc intake can result in copper deficiency and profound neurologic disease. However, when hyperzincemia is identified, the source often remains elusive. We identified four patients, one previously reported, with various neurologic abnormalities in the setting of hypocupremia and hyperzincemia. Each of these patients wore dentures and used very large amounts of denture cream chronically. Objective: To determine zinc concentration in the denture creams used by the… Expand
Zinc poisoning from excessive denture fixative use masquerading as myelopolyneuropathy and hypocupraemia
TLDR
Questioning a patient about their denture fixative usage and checking if zinc is an ingredient may be considered during an investigation for myelopolyneuropathy when vitamin B12 deficiency is not a cause. Expand
Copper Deficiency leading to Hematological and Neurological Dysfunction caused by Consumption of Zinc-containing Denture Adhesives.
TLDR
The purpose of this article is the review of the literature that documents the systemic adverse effect of excessive use of zinc-containing denture adhesive. Expand
When metals compete: a case of copper-deficiency myeloneuropathy and anemia
Background A 47-year-old woman with an otherwise unremarkable medical history was referred to the multiple sclerosis clinic by her primary neurologist for evaluation of a 2–3 year history ofExpand
Zinc-containing denture adhesive: a potential source of excess zinc resulting in copper deficiency myelopathy
TLDR
A case of a 58-year-old man diagnosed with copper deficiency myelopathy possibly due to zinc-containing denture cream overuse is presented. Expand
Zinc Burden Evokes Copper Deficiency in the Hypoalbuminemic Hemodialysis Patients
TLDR
Hemodialysis patients exhibited a lower level of zinc concentration compared to normal healthy subjects, and administration of oral zinc acetate could increase a risk for copper deficiency, so it might be better to check both zinc and copper values monthly after prescribing zinc acetates. Expand
Hyperzincemia from ingestion of denture adhesives.
The purpose of this article is to review the recent literature that documents the serious adverse systemic effects of prolonged, excessive zinc ingestion from the overuse of denture adhesives. ThisExpand
Neurological Impact of Zinc Excess and Deficiency In vivo
Zinc is an essential mineral that can cause pathological effects whether in excess or deficiency. Zinc is a component for over 250 enzymes and is required for cell growth, cell division, and cellExpand
Neurological Impact of Zinc Excess and Deficiency In vivo
Zinc is an essential mineral that can cause pathological effects whether in excess or deficiency. Zinc is a component for over 250 enzymes and is required for cell growth, cell division, and cellExpand
The predictive value of low plasma copper and high plasma zinc in detecting zinc-induced copper deficiency
TLDR
The combination of a low plasma copper and high plasma zinc is strongly predictive for the diagnosis of zinc-induced copper deficiency and therefore an opportunity for the reporting biochemist to facilitate its earlier diagnosis so enabling treatment to be implemented before the condition deteriorates. Expand
The role of the clinical biochemist in detection of zinc-induced copper deficiency
TLDR
A middle-aged woman with neutropenia and ataxia was found to have raised plasma zinc and profoundly low plasma copper concentrations and after the zinc prescription was stopped, her copper and zinc concentrations and neutropy normalized but she only had partial improvement in neurological status. Expand
...
1
2
3
4
5
...

References

SHOWING 1-10 OF 48 REFERENCES
Copper deficiency myelopathy (human swayback).
TLDR
Patients with copper deficiency myelopathy seen at the Mayo Clinic in Rochester, Minn, are described and the literature on neurological manifestations of acquired copper deficiency in humans is reviewed. Expand
Zinc-induced copper deficiency.
TLDR
The data suggest that the elimination of excess zinc is slow and that, until such elimination occurs, the intestinal absorption of copper is blocked. Expand
Zinc‐induced sideroblastic anemia: Report of a case, review of the literature, and description of the hematologic syndrome
TLDR
Hematologists should be aware of this form of reversible sideroblastic anemia, which is totally reversible with cessation of zinc intake, and appears to be zinc‐induced copper deficiency. Expand
Copper deficiency myelopathy produces a clinical picture like subacute combined degeneration
TLDR
Unrecognized copper deficiency appears to be a common cause of idiopathic myelopathy in adults, and early recognition and copper supplementation may prevent neurologic deterioration. Expand
Zinc-induced copper deficiency: a report of three cases initially recognized on bone marrow examination.
TLDR
The morphologic findings in bone marrow, while not pathognomonic, are sufficiently characteristic to suggest the diagnosis of copper deficiency, leading to further testing to establish the correct diagnosis. Expand
Oral zinc therapy for Wilson's disease.
TLDR
Oral zinc therapy, used according to the regimen, may now be considered in the treatment of patients with penicillamine intolerance, however, it is premature to convert patients to zinc therapy if they tolerate penicillsamine well. Expand
Pancytopenia after removal of copper from total parenteral nutrition.
TLDR
This is the first report of pancytopenia secondary to TPN-related copper deficiency in which the association was confirmed when hypocupremia recurred and copper supplementation was discontinued. Expand
A neurological and hematological syndrome associated with zinc excess and copper deficiency
TLDR
A patient with zinc excess and copper deficiency is reported, with the characteristic hematological syndrome of copper deficiency and a neurological syndrome with an uncommon combination of a myelopathy and generalized lower motor neuron involvement. Expand
Excessive zinc ingestion. A reversible cause of sideroblastic anemia and bone marrow depression.
TLDR
Two patients with sideroblastic anemia secondary to zinc-induced copper deficiency absorbed excess zinc secondary to oral ingestion using a zinc supplement or coins as the source. Expand
Myelopolyneuropathy and pancytopenia due to copper deficiency and high zinc levels of unknown origin: further support for existence of a new zinc overload syndrome.
TLDR
Persistent hyperzincemia without an identifiable external source appears to be a primary metabolic defect, while copper deficiency is a secondary phenomenon, causing hematologic and neurologic abnormalities, suggesting the existence of a new metabolic disorder with idiopathic zinc overload. Expand
...
1
2
3
4
5
...